Fast Method to Determine the Structural Defect Density of 156 x 156 mm2 Mc-Si Wafers
نویسندگان
چکیده
منابع مشابه
NpgRJ_Nm_1538 156..163
Degenerative and inflammatory joint diseases lead to a destruction of the joint architecture. Whereas degenerative osteoarthritis results in the formation of new bone, rheumatoid arthritis leads to bone resorption. The molecular basis of these different patterns of joint disease is unknown. By inhibiting Dickkopf-1 (DKK-1), a regulatory molecule of the Wnt pathway, we were able to reverse the b...
متن کامل156 Parrott { Integration First ?
Although the idea of teaching integration before di erentiation is certainly not new, most, if not all students are introduced to di erentiation rst. This is certainly the case in our state where many students leave school believing that the de nite integral is an application of antidi erentiation. Primarily for this reason we introduce the de nite integral in our main rst year calculus subject...
متن کامل156 Selections Reprinted From
MR0199184 (33 #7333) 14.18 Hironaka, Heisuke Resolution of singularities of an algebraic variety over a field of characteristic zero. I, II. Annals of Mathematics. Second Series 79 (1964), 109–203; ibid. (2) 79 (1964), 205–326. Outre la solution d’un problème célèbre, ce travail donne un des outils les plus puissants de la géométrie algébrique. Ce résultat est atteint avec une surprenante écono...
متن کامل156 - Hypoglycemic Agent Overdose
The primary metabolic substrate for the central nervous system (CNS) is glucose. Usual sources of glucose are diet, endogenous production (through gluconeogenesis), and storage (through glycogenolysis). Serum glucose concentrations are relatively tightly controlled by physiologic mechanisms. After dietary sources of glucose are completely used, glycogenolysis is the major physiologic mechanism ...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
ژورنال
عنوان ژورنال: Energy Procedia
سال: 2012
ISSN: 1876-6102
DOI: 10.1016/j.egypro.2012.07.048